Obesidad, insulinorresistencia, dislipemia e hipertensión: síndrome dismetabólicoPredisposing factors for type 2 diabetes and cardiovascular risk in childhood. G. Marañón. La obesidad desde el punto de vista de su pronóstico y tratamiento Consenso SEED O’ para la evaluacion del sobrepeso y la obesidad y el. La dislipemia, hipertensión, insulinorresistencia, alteración hidrocarbonada y obesidad son potentes factores de riesgo de la enfermedad cardiovascular en el .

Author: Doukree Taujin
Country: Martinique
Language: English (Spanish)
Genre: Environment
Published (Last): 23 November 2006
Pages: 38
PDF File Size: 11.39 Mb
ePub File Size: 6.97 Mb
ISBN: 805-6-32344-117-2
Downloads: 15242
Price: Free* [*Free Regsitration Required]
Uploader: Shaktitaxe

Use of fibrates in the metabolic syndrome: J Clin Invest, 95pp. Pathophysiology and pathogenesis of visceral fat obesity.

There was a problem providing the content you requested

Long-term body weight variability is ijsulinorresistencia with elevated C-reactive protein independent of current body mass index among Japanese men.

Nuevos problemas de las secreciones internas, pp. Obesity and insulin resistance in man: J Clin Endocrinol Metab, 89pp. Regulation and possible significance of leptin in humans: Metabolism, 41pp.

Insulin resistance and cardiovascular risk in the pediatric patient. Recurrence of nonalcoholic steatohepatitis following liver transplantation.


Obesidad, insulinorresistencia e hipertensión durante el embarazo

Am J Cardiol, 70pp. J Clin Endocrinol Metab, 45pp. The diverse associations of uric acid with low-grade inflammation, adiponectin and arterial stiffness in never-treated hypertensives. Enhanced expression of PAI-1 in visceral fat: J Mol Cell Biol ;8: Protein Pept Lett ; Diabetes, 17pp.

Novel modulator for endothelial adhesion molecules: Caloric restriction stimulates revascularization in response to ischemia via adiponectin-mediated activation of obessidad nitricoxide synthase. Papel del factor de necrosis tumoral en el control de las reservas grasas y la obesidad.

Positional cloning of the mouse and its human homologue. SJR uses a similar algorithm as the Google page rank; it provides a quantitative and qualitative measure of the journal’s impact.

Med Clin Barc, pp. AdipoRon, the first orally active adiponectin receptor activator, attenuates postischemic myocardial apoptosis through both AMPK-mediated and AMPK-independent signalings. World J Diabetes ;7: La grasa visceral se ha reconocido como un factor agravante en la diabetes, que se correlaciona bien con el papel del TA perivascular en la enfermedad.

Several methods have been devised to assess insulin resistance: J Vasc Res ; Best Pract Res Clin Gastroenterol, 16pp. Evidence for the occurrence of a fast acting inhibitor for tissue type plasminogen activator in plasma. Sven Med Tidskr, 5pp. Obes Res,pp.


A multifaceted syndrome responsible for NIDDM, obesity, hipertension, dyslipidemia and atherosclerotic cardiovascular disease. Trends Endocrinol Metab ; Diabetes Care, 19pp.

Adiponectin inhibits tissue factor expression and enhances tissue factor pathway inhibitor expression in human endothelial cells.

Int Obes, insulinorresistebciapp. J Clin Invest, 92pp. Adipose tissue resistin expression is severely suppressed in obesity and stimulated by peroxisoma proliferator. Estimates in vivo insulin action in man: Insulin resistance, compensatory hyperinsulinemia, and coronary heart disease. Reduced oxygenation in human obese adipose tissue is associated with impaired insulin suppression of lipolysis.

Arterioscler Thromb Vasc Biol, 20pp. Prospective studies of Pima Indians. Diab Metabol Ovesidad, 14pp.

Macrophage activation is responsible for loss of anticontractile function in inflamed perivascular fat. Ndahimana D, Kim EK.